These data indicate that NPM/ALK activates STAT3 and that PP2A and lack of protein inhibitor of activated STAT3 may be important in maintaining STAT3 in the activated state in the ALK+ TCL cells.     ///    Here we report that NPM/ALK induces continuous activation of STAT3.     ///    "Downstream effector molecules triggered by NPM/ALK remain, however, largely unidentified. "    ///    "Furthermore, STAT3 was constitutively associated with NPM/ALK in the ALK+ TCL cell lines. "    ///    Transfection of BaF3 cells with NPM/ALK resulted in tyrosine phosphorylation of STAT3. 