In t(8;21) leukaemic cells expressing the aberrant fusion protein AML1-ETO, we demonstrate that this protein is part of a transcription factor complex binding to extended sequences of the c-FMS intronic regulatory region rather than the promoter.     ///    "The AML1-ETO complex does not disrupt binding of other transcription factors, indicating that c-FMS is not irreversibly epigenetically silenced. "    ///    "However, AML1-ETO binding correlates with changes in the histone modification pattern and increased association of histone deacetylases. "    ///    Epigenetic consequences of AML1-ETO action at the human c-FMS locus.. 