p53 signaling in response to increased DNA damage sensitizes AML1-ETO cells to stress-induced death..     ///    We thus speculate that AML1-ETO may facilitate accumulation of genetic alterations by suppressing endogenous DNA repair.     ///    "Expression of the AML1-ETO (AE) fusion protein alone is not sufficient to induce leukemia, but the nature of the additional genetic alterations is unknown. "