Identification of additional cytogenetic and molecular genetic abnormalities in acute myeloid leukaemia with t(8;21)/AML1-ETO..     ///    We analysed 99 patients with an AML1-ETO rearrangement for additional aberrations.     ///    "These clinical findings support the model that AML1-ETO collaborates with other genetic alterations, such as mutations of receptor tyrosine kinases, to induce AML.. "    ///     AML1-ETO collaborates with further genetic abnormalities to induce acute myeloid leukaemia (AML). 