A novel patient-derived tumorgraft model with TRAF1-ALK anaplastic large-cell lymphoma translocation..     ///    These data indicate that the activation of NFkB signaling contributes to the neoplastic phenotype of TRAF1-ALK ALCL.     ///    The treatment with proteasome inhibitors of TRAF1-ALK cells led to the downregulation of p50/p52 and lymphoma growth inhibition. 